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Reflections on B Cells in SLE & Lupus Nephritis

Ruth Jessen Hickman, MD  |  December 5, 2025

Looking forward, Dr. Jackson stated that he believes that the field will explore multiple avenues of CAR T cell therapy. Initial work has used autologous CAR T cells, but Dr. Jackson pointed out the potential utility of using allogenic CAR T cells. Scientists are also pursuing such work as engineering natural killer cells instead of T cells (CAR NK), generating CAR T cells and NK cells from induced pluripotent stem cells, finding ways to deliver CAR T in vivo (without removal of the patient’s T cells) and using T cell engagers, a kind of bispecific monoclonal antibody.

Dr. Jackson said that most CAR T cell trials in SLE focus on CD19 as a target. However, his lab is also exploring B cell maturation antigen (BCMA), a marker that is highly expressed on plasma cells. For certain autoimmune diseases, targeting BCMA might be important to deplete pathogenic long-lived plasma cells in the bone marrow, which are not eliminated by CD19-directed therapies.

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Dr. Jackson emphasized that although identifying the optimal cell target must be validated in human clinical trials, the next decade promises exciting advances in the treatment of lupus and other humoral autoimmune diseases. Researchers aiming to change the paradigm for these diseases with CAR therapies will need to explore not just different modalities, but also alternative targets like BCMA, he urged.


Ruth Jessen Hickman, MD, a graduate of the Indiana University School of Medicine, is a medical and science writer in Bloomington, Ind.

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References

  1. Rovin BH, Furie R, Latinis K, et al. Efficacy and safety of rituximab in patients with active proliferative lupus nephritis: The Lupus Nephritis Assessment with Rituximab study. Arthritis Rheum. 2012 Apr;64(4):1215–1226.
  2. Mo S, Li Y, He J, Lin L. Progress of rituximab in the treatment of systemic lupus erythematosus and lupus nephritis. Front Med (Lausanne). 2024 Oct 4;11:1472019.
  3. Schett G, Mackensen A, Mougiakakos D. CAR T-cell therapy in autoimmune diseases. Lancet. 2023 Nov 25;402(10416):2034–2044.
  4. Mackensen A, Müller F, Mougiakakos D, et al. Anti-CD19 CAR T cell therapy for refractory systemic lupus erythematosus. Nat Med. 2022 Oct;28(10):2124–2132.
  5. Jackson SW, Scharping NE, Kolhatkar NS, et al. Opposing impact of B cell-intrinsic TLR7 and TLR9 signals on autoantibody repertoire and systemic inflammation. J Immunol. 2014 May 15;192(10):4525–4532.
  6. Jackson SW, Jacobs HM, Arkatkar T, et al. B cell IFN-γ receptor signaling promotes autoimmune germinal centers via cell-intrinsic induction of BCL-6. J Exp Med. 2016 May 2;213(5):733–750.
  7. Arkatkar T, Du SW, Jacobs HM, et al. B cell-derived IL-6 initiates spontaneous germinal center formation during systemic autoimmunity. J Exp Med. 2017 Nov 6;214(11):3207–3217.
  8. Liu S, Lagos J, Shumlak NM, et al. NADPH oxidase exerts a B cell-intrinsic contribution to lupus risk by modulating endosomal TLR signals. J Exp Med. 2024 Apr 1;221(4):e20230774.
  9. Gordon RA, Cosgrove HA, Marinov A, et al. NADPH oxidase in B cells and macrophages protects against murine lupus by regulation of TLR7. JCI Insight. 2024 Jul 23;9(16):e178563.
  10. Danaher P, Hasle N, Nguyen ED, et al. Childhood-onset lupus nephritis is characterized by complex interactions between kidney stroma and infiltrating immune cells. Sci Transl Med. 2024 Nov 27;16(775):eadl1666.

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